Download Andrology: Male Reproductive Health and Dysfunction by Eberhard Nieschlag (auth.), Eberhard Nieschlag, Hermann M. PDF

By Eberhard Nieschlag (auth.), Eberhard Nieschlag, Hermann M. Behre, Susan Nieschlag (eds.)

ISBN-10: 3540783547

ISBN-13: 9783540783541

This is the 3rd, completely revised variation of a well known textbook that keeps to symbolize the greatest within the literature on scientific andrology. It examines extensive all points of male reproductive future health, encompassing the elemental body structure of male reproductive functionality and a variety of problems. all of the chapters is written through notable specialists within the box. one of the subject matters coated are:

  • Testicular function
  • Sperm maturation and fertilization
  • Diagnostic techniques
  • Infertility
  • Primary and secondary hypogonadism
  • Late-onset hypogonadism
  • Erectile dysfunction
  • Genetic disorders
  • Environmental influences
  • Psychology and sexual medicine
  • Testosterone therapy
  • Assisted copy: TESE and ICSI
  • Cryopreservation
  • Male contraception
  • Ethics in andrology

This quantity will function a textbook for the newcomer to andrology, whereas delivering the skilled health practitioner with a important reference paintings. it's going to attract all who're considering male reproductive wellbeing and fitness:

  • Andrologists
  • Urologists
  • Endocrinologists
  • Gynecologists
  • Internists
  • Pediatricians
  • Basic scientists

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Additional info for Andrology: Male Reproductive Health and Dysfunction

Sample text

Both Sertoli cell number and expression of markers of cell division are stimulated by these hormones. The division of Sertoli cells ends when the first germ cells undergo meiotic division and Sertoli cells have built tight junctions between each other, the 16 so-called blood-testis-barrier (see Sect. 5). Lack of connexin-43, a predominant gap-junction protein, prevents Sertoli cell maturation associated with continued division of Sertoli cells and spermatogenic arrest beyond spermatogonial development (Brehm et al.

CAMP remains, therefore, the main signal transducer and calcium could possibly act as a signal amplification or modulating mechanism. Following the hormone-receptor interaction there is an increase in cAMP concentrations and subsequent activation of protein kinaseses which, in turn, phosphorylate existing proteins such as enzymes, structural and transport proteins and transcriptional activators. Activating and inactivating mutations of the gonadotropin receptors have been identified. The biological consequences of these mutations are described below and in Chap.

1 Structure of Gonadotropins LH and FSH are glycoprotein hormones secreted by the pituitary gland that control development, maturation and function of the gonad. Like the related thyroid stimulating hormone (TSH) and human chorionic gonadotropin (hCG), they consist of two polypeptide chains, α and β, bearing carbohydrate moieties N-linked to asparagine (Asn) residues. The α subunit is common to all members of the glycoprotein G. F. Weinbauer et al. hormone family, whereas the β subunit, although structurally very similar, differs in each hormone and confers specificity of action.

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